Photon 333 donated @Photon
03 April, 01:03
@TheMac Here's your challenge.

There are anecdotal stories of Benadryl working for C19. Quinine, HCQ and Ivermectin. All of those reduce acetylcholine and, therefore, muscarinic and nicotinic receptor activity of organs.

Do they keep C19 from binding to organs? Is that the key to preventing C19? Do they disable the ability of C19 to bind to ACE2 receptors?

I was going to be a biochemist. [They] have biochemists working on these diabolical plots. So we need to look at mechanisms in the body. Thanks.

https://www.frontiersin.or...

https://www.the-scientist....

https://pubmed.ncbi.nlm.ni...

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Photon 333 donated @Photon
03 April, 01:08
In response Photon 333 to her Publication
@TheMac Pt 2

Marijuana does the opposite of those supplements/drugs. Pot increases acetylcholine. Is that why the bastards are trying to legalize pot? So they can kill more unsuspecting people, who they believe are too dumb to make these connections?

And what if they alter the marijuana itself?

Don't be fooled, everyone. They are already changing bodies via air, food, and water. Sinister bastards.

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Photon 333 @Photon
Caffeine increases acetylcholine as well. Caffeine is a cholinesterase inhibitor.

Acetylcholine (ACh) regulates, for example, whether a muscle works well or not. If you don't have enough of what I call "muscle gas," your muscles won't work well.

If you've had an anesthetic, you can easily understand this. Anesthetics reduce acetylcholine too. Can you move or breathe (or think) well after having anesthesia? Nope.

Anesthetics, HCQ, Quinine, Benadryl, Ivermection, Atropine, and many other foods and drugs can reduce ACh.

Marijuana, Mestinon, caffeine, etc. increase ACh.

[THEY] will do anything they can to harm us.
01:39 PM - Apr 03, 2022
In response Photon 333 to her Publication
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